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By Ricardo Tapia (auth.), Leslie L. Iversen, Susan D. Iversen, Solomon H. Snyder (eds.)

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R, and JABBUR, S. , 1971, The effects of depleting GABA on cuneate presynaptic inhibition, Brain Res. 33:530-532. BARKER, J. , and NICOLL, R. , 1972, Gamma-aminobutyric acid: Role in primary afferent depolarization, Science 176: 1043-1045. BAXTER, C. , 1969, Changes in gamma-aminobutyric acid-shunt enzymes and substrates after administration of carbonyl reagents and vitamin B6 in vivo: An apparent discrepancy in assay techniques, Ann. N. Y. Acad. Sci. 166:267-280. BAXTER, C. , 1970, The nature of y-aminobutyric acid, in: Handbook of Neurochemistry, Vol.

It has been suggested that defects of the GABA system are also involved in schizophrenia (Roberts, 1972). Our present knowledge of GABA metabolism and the characteristics of the activity of the participating enzymes is relatively advanced. Much less understood are the membrane phenomena responsible for the release and the uptake of GABA and the mechanisms by which GABA interacts with its receptor and hyperpolarizes the postsynaptic membrane. With this knowledge and the new information that will certainly be gained on the still obscure mechanisms of how the GABA system works, the future of advances in the pharmacology of GABA and its applications to prevent, ameliorate, or even cure some of the most dreadful neurological diseases look very promising.

Much less understood are the membrane phenomena responsible for the release and the uptake of GABA and the mechanisms by which GABA interacts with its receptor and hyperpolarizes the postsynaptic membrane. With this knowledge and the new information that will certainly be gained on the still obscure mechanisms of how the GABA system works, the future of advances in the pharmacology of GABA and its applications to prevent, ameliorate, or even cure some of the most dreadful neurological diseases look very promising.

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